UNDERSTANDING OF CEREBROVASCULAR DISEASE WITH AN EMPHASIS ON BRAIN STROKE IN THE POPULATION OF KOLKATA- A STUDY
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Keywords
Cerebrovascular disease, stroke, health, awareness, risk
Abstract
Over the past few decades, Cerebrovascular diseases (CVDs) have been responsible for a significant number of deaths in India. The age-wise global data of stroke incidence indicates that India already has a higher proportion compared to global standards. The CVD epidemic in India is a major cause for concern due to its accelerated build-up, the early onset of disease in the population, and the high case fatality rate. The epidemiological transition from infectious to non-communicable diseases in India has occurred rapidly. Premature mortality, in terms of years of life lost, due to CVDs in India has increased by 59% from 23.2 million (1990) to 37 million (2010). This is alarming, particularly given the early age of onset of the disease. To assess the awareness of CVDs among the population of Kolkata, a survey was conducted with an emphasis on college and university-going students. The survey was conducted using a questionnaire-based approach, with 75 participants voluntarily taking part over a six-week period in June and July 2022. To understand the level of awareness and perception about CVDs among the participants, data visualization methods and descriptive statistics were used. The results highlighted a significant lack of knowledge about CVDs among the participants. Findings showed 41% of the participants were unaware of the different types of brain strokes, and 48.5% of the population had scattered and unclear knowledge about the symptoms of the disease and the correlation of cerebrovascular disease with hypertension. Conversely, 85.5% participants responded correctly towards the recovery from brain strokes. On the same pitch, 74.7% of the respondents were aware about smoking as the predisposing factor for brain stroke, whereas 80% of the volunteers answered right about the extent of damage caused by the strokes. The study suggests that there is an urgent need for increased education and awareness campaigns about stroke and its risk factors in the population. Awareness, regular campaigns could help to increase the chances of early detection and treatment of stroke, which could ultimately lead to better outcomes for stroke patients in and around Kolkata.
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1. Anderson JJ, Baron G, van der Heijde D, Felson DT, Dougados M. Ankylosing Spondylitis Assessment Group preliminary definition of short-lerm improvement in ankylosing spondylitis. Arthritis Rheum 2001;44:1876-86. 2. Arends S, Lebbink HR, Spoorenberg A, et al. The formation of autoantibodies and antibodies to TNF-et blocking agents in relation to clinical response in patients with ankylosing spondylitis. Clin Exp Rheumatol 2010;28:661-8. 3. Bal A, Unlu E, Bahar G, et al. Comparison of serum IL-1 beta, slL-2R, IL-6, and TNF-alpha levels with disease activity parameters in ankylosing spondylitis. Clin Rheumatol 2007;26:211-15. 4. Baraliakos X, Listing J, Rudwaleit M, et al. The relationship between infiammation and new bone formation in patients with ankylosing spondylitis. Arthritis Res Ther 2008; 10:R104. 5. Brandt J, Listing J, Sieper J, Rudwaleit M, van der Heijde D, Braun J. Development and preselection of criteria for short term improvement after anti-TNFα treatment in ankylosing spondylitis .Ann Rheum Dis 2004;63:1438-44. 6. Calin A, Garrett S, Whitelock H, et al. A new approach to defining functional ability in ankylosing spondylitis: the development of the Bath Ankylosing Spondylicis Functional Index. J Rheumatol 1994;21(12):2281-2285. 7. Calin A, Nakache JP, Gueguen A, Zeidler H, Mielants H, Dougados M. Defining disease activity in ankylosing spondylitis: is c combination of variables (Bath Ankylosing Spondylitis Disease Activity Index) an appropriate instrument? Rheumatology (Oxford) 1999;38:878-82. 8. Chen J, Liu C. Sulfasalazine for ankylosing spondylitis. Cochrane Database Syst Rev 2005;(2):CD004800. 9. Conti F, Ceccarelli F, Marocchi E, Magrini L, Spinelli FR, Spadaro A, et al. Switching tumour necrosis factor α antagonists in patients with ankylosing spondylitis and psoriatic arthritis: an observational study over a 5-year period. Ann Rheum Dis 2007;66:1393-7. 10. de Vries MK, van Eijk IC, van der Horst-Eruinsma IE, et al. Erythrocyte sedimentation rate, C-reactive protein level, and serum amyloid a protein for patient selection and monitoring of anti-tumor necrosis factor treatment in ankylosing spondylitis. Arthritis Rheum 2009;61:1484-90. 11. Dougados M, Boumer P. Amor B. Sulphosalazine in ankylosing spondylitis: a double blind controlled study in 60 patients. BMJ 1986;293:911-14. 12. Dougados M, van der Linden S, Leirisalo-Repo M, Huitfeldt B, Juhlin R, Veys E, et al. Sulfasalazine in the treatment of spondyloarthropathy. A randomized, multicenter, double-blind, placebo-controlled study. Arthritis Rheum 1995;38:618-27. 13. Gossec L, van der Heijde D. Melian A, Krupa DA, James MK, Cavanaugh Jr PF, et al. The efficacy of cyclooxygenase-2 inhibition by etoricoxib and naproxen on the axial manifestations of ankylosing spondylitis in the presence of peripheral arthritis. Ann Rheum Dis 2005;62:1563-7. 14. Heuft-Dorenbosch L, van Tubergen A, Spoorenberg A, Landewe R, Dougados M, Mielants H, et al. The influence of peripheral arthritis on disease activity in ankylosing spondylitis patients as measured with the Bath Ankylosing Spondylitis Disease Activity Index. Arthritis Rheum2004;51:154-9. 15. Jenkinson TR, Mallorie PA, Whitelock HC, Kennedy LG, Garrett SL, Calin A. Defining spinal mobility in ankylosing spondylities (AS): the Bath AS Metrology Index. J Rheumatol 1994;21:1694-8. 16. Jones SD, Calin A, Steiner A. An update on the Bath Ankylosing Spondylitis Disease Activity and Functional Indices (BASDÁI, BASFI): excellent Cronbach's alpha scores. J Rheumatol 1996;23:407. 17. Maksymowych WP, Landewé R. Conner-Spady B, et al. Serum Matrix metalloproteinase 3 is an independent predictor of structural darnage progression in patients with ankylosing spondylitis. Arthritis Rheum 2007;56:1846-53. 18. Nissila M. Lehtinen K Leirisalo-Repo M, Luukkainen R, Mutru O, Yli-Kerttula U. MSulfasalazine in the treatment of ankylosing spondylitis. A twenty-six-week, placebo-controlled clinical trial. Arthritis Rheum 1988;31:1111-16. 19. Park MC, Lee SW, Choi ST, et al. Serum leptin levels correlate with interleukin-6 levels and disease activity in patients with ankylosing spondylitis. Scand J Rheumatol 2007;36:101-6. 20. Rudwaleit M, Schwarzlose S, Hilgert ES, Listing J, Sieper J. Magnetic resonance imaging (MRI) in predicting a major clinical responce to anti-TNF-treatment in ankylosing spondylitis.Ann Rheum Dis 2007.E-pub ahead of print. 21. Rudwaleit M. Schwarzlose S, Hilgert ES, Listing J, Braun ), Sieper J. MRI in predicting a major clinical response to anti-tumour necrosis factor treatment in ankylosing spondylitis. Ann Rheum Dis 2008;67:126-81. 22. Spoorenberg A, van der Heijde D, de Klerk E, Dougados M, de Vlam K, Mielants H et al. Relative value of erythrocyte sedimentation rate and C-reactive protein in assessment of disease activity in ankylosing spondylitis. J Rheumatol 1999;26:980-4. 23. Van der Heijde D, Landewe R, Feldtkeller E. Proposal of a linear definition of the Bath Ankylosing spondylitis Metrology Index (BASMI) and comparison with the 2-step and 10-step definitions.Ann Rheum Dis 2008;67:489-93. 24. Visvanathan S, Wagner C, Marini JC, et al. Inflammatory biomarkers, disease activity and spinal disease measures in patients with ankylosing spondylities after treatment with infliximab. Ann Rheum Dis 2008; 67:511-517. 25. Yang C, Gu J. Rihl M, et al. Serum levels of matrix metalloproteinase 3 and macrophage colony-stimulating factor 1 correlate with disease activity in ankylosing spondylitis. Arthritis Rheum 2004:51:691-9.
